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dc.contributor.authorda Silveira, SA
dc.contributor.authorKikuchi, S
dc.contributor.authorFossati-Jimack, L
dc.contributor.authorMoll, T
dc.contributor.authorSaito, T
dc.contributor.authorVerbeek, JS
dc.contributor.authorBotto, M
dc.contributor.authorWalport, MJ
dc.contributor.authorCarroll, M
dc.contributor.authorIzui, S
dc.date.accessioned2021-01-20T12:57:57Z
dc.date.available2021-01-20T12:57:57Z
dc.date.issued2002-03-18
dc.identifier.citationSamareh Azeredo da Silveira, Shuichi Kikuchi, Liliane Fossati-Jimack, Thomas Moll, Takashi Saito, J. Sjef Verbeek, Marina Botto, Mark J. Walport, Michael Carroll, Shozo Izui; Complement Activation Selectively Potentiates the Pathogenicity of the IgG2b and IgG3 Isotypes of a High Affinity Anti-Erythrocyte Autoantibody . J Exp Med 18 March 2002; 195 (6): 665–672. doi: https://doi.org/10.1084/jem.20012024en_US
dc.identifier.issn0022-1007
dc.identifier.urihttps://qmro.qmul.ac.uk/xmlui/handle/123456789/69869
dc.format.extent665 - 672
dc.publisherRockefeller University Pressen_US
dc.relation.ispartofJournal of Experimental Medicine
dc.subjectautoimmune hemolytic anemiaen_US
dc.subjectcomplement receptoren_US
dc.subjectFc receptoren_US
dc.subjectIgG isotypeen_US
dc.subjectphagocytosisen_US
dc.subjectAUTOIMMUNE HEMOLYTIC-ANEMIAen_US
dc.subjectREVERSE ARTHUS REACTIONen_US
dc.subjectGAMMA-RIII CD16en_US
dc.subjectFC-RECEPTORSen_US
dc.subjectIN-VIVOen_US
dc.subjectDEFICIENT MICEen_US
dc.subjectCHAIN GENEen_US
dc.subjectINFLAMMATORY RESPONSESen_US
dc.subjectMOUSE MACROPHAGESen_US
dc.subjectGLOMERULONEPHRITISen_US
dc.titleComplement activation selectively potentiates the pathogenicity of the IgG2b and IgG3 Isotypes of a high affinity anti-erythrocyte autoantibodyen_US
dc.typeArticleen_US
dc.rights.holder(c) The Rockefeller University Press
dc.identifier.doihttps://doi.org/10.1084/jem.20012024
pubs.issue6en_US
pubs.notesNot knownen_US
pubs.volume195en_US
rioxxterms.funderDefault funderen_US
rioxxterms.identifier.projectDefault projecten_US


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