| dc.contributor.author | Schroth, J | en_US |
| dc.contributor.author | Henson, SM | en_US |
| dc.date.accessioned | 2020-11-11T08:30:20Z | |
| dc.date.available | 2020-10-14 | en_US |
| dc.date.issued | 2020-10-16 | en_US |
| dc.identifier.issn | 2084-6835 | en_US |
| dc.identifier.uri | https://qmro.qmul.ac.uk/xmlui/handle/123456789/68158 | |
| dc.description.abstract | We review here the seminal findings of Desdin-Mico et al. showing that T cells with dysfunctional mitochondria induce multimorbity and premature senescence, due to mitochondrial transcription factor A (TFAM). They add further weight to the idea that targeting immunometabolism could be beneficial in combating the detrimental effects of age-related disease. | en_US |
| dc.format.extent | e200035 - ? | en_US |
| dc.language | eng | en_US |
| dc.language.iso | en | en_US |
| dc.relation.ispartof | Immunometabolism | en_US |
| dc.subject | T cell | en_US |
| dc.subject | ageing | en_US |
| dc.subject | metabolism | en_US |
| dc.subject | mitochondria | en_US |
| dc.subject | senescence | en_US |
| dc.title | Mitochondrial Dysfunction Accelerates Ageing. | en_US |
| dc.type | Article | |
| dc.identifier.doi | 10.20900/immunometab20200035 | en_US |
| pubs.author-url | https://www.ncbi.nlm.nih.gov/pubmed/33101729 | en_US |
| pubs.issue | 4 | en_US |
| pubs.notes | Not known | en_US |
| pubs.publication-status | Published online | en_US |
| pubs.volume | 2 | en_US |
| rioxxterms.funder | Default funder | en_US |
| rioxxterms.identifier.project | Default project | en_US |
