The pathogenic role of Stat2 in inflammation is independent of canonical interferon signaling and is mediated by TNFa
dc.contributor.author | Heath, H | en_US |
dc.contributor.author | Kudo, H | en_US |
dc.contributor.author | Renney, G | en_US |
dc.contributor.author | Ward, M | en_US |
dc.contributor.author | Cutillas, P | en_US |
dc.contributor.author | Goldin, RD | en_US |
dc.contributor.author | Hutchins, R | en_US |
dc.contributor.author | Foster, G | en_US |
dc.contributor.author | Alazawi, W | en_US |
dc.date.accessioned | 2020-08-11T15:46:23Z | |
dc.date.issued | 2018-04 | en_US |
dc.identifier.issn | 0168-8278 | en_US |
dc.identifier.uri | https://qmro.qmul.ac.uk/xmlui/handle/123456789/66214 | |
dc.format.extent | S612 - S612 | en_US |
dc.title | The pathogenic role of Stat2 in inflammation is independent of canonical interferon signaling and is mediated by TNFa | en_US |
dc.type | Conference Proceeding | |
dc.identifier.doi | 10.1016/S0168-8278(18)31480-6 | en_US |
pubs.author-url | http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000461068603023&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6a | en_US |
pubs.notes | Not known | en_US |
pubs.publication-status | Published | en_US |
pubs.volume | 68 | en_US |
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Centre for Immunobiology [1121]