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dc.contributor.authorMiess, Hen_US
dc.contributor.authorDankworth, Ben_US
dc.contributor.authorGouw, AMen_US
dc.contributor.authorRosenfeldt, Men_US
dc.contributor.authorSchmitz, Wen_US
dc.contributor.authorJiang, Men_US
dc.contributor.authorSaunders, Ben_US
dc.contributor.authorHowell, Men_US
dc.contributor.authorDownward, Jen_US
dc.contributor.authorFelsher, DWen_US
dc.contributor.authorPeck, Ben_US
dc.contributor.authorSchulze, Aen_US
dc.date.accessioned2020-01-22T13:10:35Z
dc.date.available2018-04-08en_US
dc.date.issued2018-10-04en_US
dc.identifier.issn0950-9232en_US
dc.identifier.urihttps://qmro.qmul.ac.uk/xmlui/handle/123456789/62485
dc.descriptionThe publisher's final edited version of this article is available at Oncogene. 2018 Oct;37(40):5435-5450. doi: 10.1038/s41388-018-0315-z. Epub 2018 Jun 5. Non-commercial use onlyen_US
dc.description.sponsorshipThis study was funded by Cancer Research UK, the German Research Foundation (FOR2314) and the German Cancer Aid (111917).en_US
dc.format.extent5435 - 5450en_US
dc.language.isoenen_US
dc.relation.ispartofONCOGENEen_US
dc.rightsMacmillan Publishers Limited, part of Springer Nature 2018
dc.titleThe glutathione redox system is essential to prevent ferroptosis caused by impaired lipid metabolism in clear cell renal cell carcinomaen_US
dc.typeArticle
dc.identifier.doi10.1038/s41388-018-0315-zen_US
pubs.author-urlhttp://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000446312900005&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6aen_US
pubs.issue40en_US
pubs.notesNot knownen_US
pubs.publication-statusPublisheden_US
pubs.volume37en_US
rioxxterms.funderDefault funderen_US
rioxxterms.identifier.projectDefault projecten_US


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