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Protein kinase C epsilon is required for macrophage activation and defense against bacterial infection

dc.contributor.authorCastrillo, Aen_US
dc.contributor.authorPennington, DJen_US
dc.contributor.authorOtto, Fen_US
dc.contributor.authorParker, PJen_US
dc.contributor.authorOwen, MJen_US
dc.contributor.authorBosca, Len_US
dc.date.accessioned2016-11-25T17:05:13Z
dc.date.issued2001-11-05en_US
dc.date.submitted2016-11-16T18:12:38.221Z
dc.identifier.issn0022-1007en_US
dc.identifier.urihttp://qmro.qmul.ac.uk/xmlui/handle/123456789/17892
dc.description.sponsorshipImperial Cancer Research Fund, and grants FEDER 2FD-1997-1432 from Comisión Interministerial de Ciencia y Tecnología (CICYT) and PM98-0120 from Dirección General de Enseñanza Superior e Investigación Científica, Spain. F. Otto was supported by Deutsche Forschungsgemeinschaft grant Ot 134/1-1.en_US
dc.format.extent1231 - 1242en_US
dc.relation.ispartofJ EXP MEDen_US
dc.rightsAfter the Initial Publication Period, RUP will grant to the public the non- exclusive right to copy, distribute, or display the Work under a Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported license as described at http://creativecommons.org/licenses/by-nc-sa/3.0/ and http://creativecommons.org/licenses/by-nc-sa/3.0/legalcode.
dc.subjectnitric oxideen_US
dc.subjectprotein kinase Cen_US
dc.subjectnitric oxide synthaseen_US
dc.subjectmacrophage activationen_US
dc.subjectbacterial infectionen_US
dc.subjectNF-KAPPA-Ben_US
dc.subjectNITRIC-OXIDE SYNTHASEen_US
dc.subjectLIPOPOLYSACCHARIDE-STIMULATED MACROPHAGESen_US
dc.subjectTUMOR-NECROSIS-FACTORen_US
dc.subjectTOLL-LIKE RECEPTOR-4en_US
dc.subjectSIGNAL-TRANSDUCTION PATHWAYen_US
dc.subjectCELL WALL COMPONENTSen_US
dc.subjectPKC-EPSILONen_US
dc.subjectINTERFERON-GAMMAen_US
dc.subjectMAMMALIAN-CELLSen_US
dc.titleProtein kinase C epsilon is required for macrophage activation and defense against bacterial infectionen_US
dc.typeArticle
dc.rights.holder(c) The Rockefeller University Press
pubs.issue9en_US
pubs.notesNot knownen_US
pubs.volume194en_US


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