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dc.contributor.authorLanna, Aen_US
dc.contributor.authorHenson, SMen_US
dc.contributor.authorEscors, Den_US
dc.contributor.authorAkbar, ANen_US
dc.date.accessioned2016-10-11T11:47:24Z
dc.date.available2014-07-29en_US
dc.date.issued2014-10en_US
dc.date.submitted2016-09-09T16:22:42.996Z
dc.identifier.issn1529-2908en_US
dc.identifier.other10.1038/ni.2981
dc.identifier.urihttp://qmro.qmul.ac.uk/xmlui/handle/123456789/15827
dc.description.sponsorshipWe thank S.S. Marelli for discussions. Supported by the Medical Research Council (A.L.), the Biotechnology and Biological Science Research Council (BB/J006750/1 to A.N.A. and S.M.H.) and the Instituto de Salud Carlos III, Spain (D.E.).en_US
dc.format.extent965 - U211en_US
dc.language.isoenen_US
dc.relation.ispartofNATURE IMMUNOLOGYen_US
dc.rightsAll rights reserved
dc.titleThe kinase p38 activated by the metabolic regulator AMPK and scaffold TAB1 drives the senescence of human T cellsen_US
dc.typeArticle
dc.rights.holder2014. Nature America
dc.identifier.doi10.1038/ni.2981en_US
pubs.author-urlhttp://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000342564800012&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6aen_US
pubs.issue10en_US
pubs.notesNot knownen_US
pubs.publication-statusPublisheden_US
pubs.volume15en_US


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