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Abstract P2005: Loss Of Full-length Mylk3 Causes Dilated Cardiomyopathy Via A MYL2-independent Mechanism

dc.contributor.authorWilliams, JLen_US
dc.contributor.authorPaudyal, Aen_US
dc.contributor.authorStewart, Men_US
dc.contributor.authorCutillas, Pen_US
dc.contributor.authorTinker, Aen_US
dc.contributor.authorMetherell, Len_US
dc.date.accessioned2022-12-08T09:07:26Z
dc.date.issued2022-08-05en_US
dc.identifier.issn0009-7330en_US
dc.identifier.urihttps://qmro.qmul.ac.uk/xmlui/handle/123456789/83018
dc.format.extentap2005 - ap2005en_US
dc.publisherWolters Kluweren_US
dc.relation.ispartofCirculation Researchen_US
dc.subjectCardiovascularen_US
dc.subjectGeneticsen_US
dc.subjectHeart Diseaseen_US
dc.subject2 Aetiologyen_US
dc.subject2.1 Biological and endogenous factorsen_US
dc.titleAbstract P2005: Loss Of Full-length Mylk3 Causes Dilated Cardiomyopathy Via A MYL2-independent Mechanismen_US
dc.typeArticle
dc.identifier.doi10.1161/res.131.suppl_1.p2005en_US
pubs.issueSuppl_1en_US
pubs.notesNot knownen_US
pubs.volume131en_US
qmul.funderThe role of MYLK3 in cardiac signalling and development::British Heart Foundationen_US
qmul.funderThe role of MYLK3 in cardiac signalling and development::British Heart Foundationen_US


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