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Novel LZTR1 mutations in subjects with features of Noonan Syndrome and GH insensitivity negatively regulate GH-induced IGF-I production and hyperactivate GH-induced ERK1/2 activation in response to GH in vitro

dc.contributor.authorChatterjee, S
dc.contributor.authorBertola, DR
dc.contributor.authorAgwu, C
dc.contributor.authorMaharaj, A
dc.contributor.authorWilliams, J
dc.contributor.authorCottrell, E
dc.contributor.authorShapiro, L
dc.contributor.authorAndrews, A
dc.contributor.authorSavage, MO
dc.contributor.authorGaston-Massuet, C
dc.contributor.authorMetherell, LA
dc.contributor.authorStorr, HL
dc.date.accessioned2021-10-21T10:29:59Z
dc.date.available2021-10-21T10:29:59Z
dc.date.issued2021-09
dc.identifier.issn1663-2818
dc.identifier.urihttps://qmro.qmul.ac.uk/xmlui/handle/123456789/74649
dc.format.extent322 - 323
dc.titleNovel LZTR1 mutations in subjects with features of Noonan Syndrome and GH insensitivity negatively regulate GH-induced IGF-I production and hyperactivate GH-induced ERK1/2 activation in response to GH in vitroen_US
dc.typeConference Proceedingen_US
pubs.author-urlhttp://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000696302600600&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6aen_US
pubs.issueSUPPL 1en_US
pubs.notesNot knownen_US
pubs.publication-statusPublisheden_US
pubs.volume94en_US
qmul.funderInvestigating the role of the sphingolipid pathway in a novel syndromic form of primary adrenal insufficiency::Barts Charityen_US
qmul.funderInvestigating the role of the sphingolipid pathway in a novel syndromic form of primary adrenal insufficiency::Barts Charityen_US


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