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Fetus-derived DLK1 is required for maternal metabolic adaptations to pregnancy and is associated with fetal growth restriction

dc.contributor.authorCleaton, MAMen_US
dc.contributor.authorDent, CLen_US
dc.contributor.authorHoward, Men_US
dc.contributor.authorCorish, JAen_US
dc.contributor.authorGutteridge, Ien_US
dc.contributor.authorSovio, Uen_US
dc.contributor.authorGaccioli, Fen_US
dc.contributor.authorTakahashi, Nen_US
dc.contributor.authorBauer, SRen_US
dc.contributor.authorCharnock-Jones, DSen_US
dc.contributor.authorPowel, TLen_US
dc.contributor.authorSmith', GCSen_US
dc.contributor.authorFerguson-Smith, ACen_US
dc.contributor.authorCharalambous, Men_US
dc.date.accessioned2016-11-04T09:38:24Z
dc.date.available2016-09-26en_US
dc.date.issued2016-12en_US
dc.date.submitted2016-10-10T17:34:29.145Z
dc.identifier.issn1061-4036en_US
dc.identifier.urihttp://qmro.qmul.ac.uk/xmlui/handle/123456789/16283
dc.description.sponsorshipMAMC was supported by a PhD studentship from the Cambridge Centre for Trophoblast Research. Research was supported by grants from the MRC (MR/J001597/1; MR/L002345/1), The Medical College of Saint Bartholomew's Hospital Trust, Wellcome Trust Investigator Award, EpigeneSys (FP7 Health - 257082), EpiHealth (FP7 Health – 278414), a Herchel Smith Fellowship (NT), NIH RO1 DK89989. Contents are the authors’ sole responsibility and do not necessarily represent official NIH views. We thank G Burton for invaluable support, M Constancia and I Sandovici (University of Cambridge, UK) for the Meox2Cre mice. We are extremely grateful to all of the participants in the Pregnancy Outcome Prediction study. This work was supported by the National Institute for Health Research (NIHR) Cambridge Comprehensive Biomedical Research Centre (Women's Health theme), and project grants from the MRC (G1100221) and Sands (Stillbirth and neonatal death charity). The study was also supported by GE Healthcare (donation of two Voluson i ultrasound systems for this study), and by the NIHR Cambridge Clinical Research Facility, where all research visits took place.en_US
dc.format.extent1473 - 1480en_US
dc.language.isoenen_US
dc.relation.ispartofNATURE GENETICSen_US
dc.titleFetus-derived DLK1 is required for maternal metabolic adaptations to pregnancy and is associated with fetal growth restrictionen_US
dc.typeArticle
dc.rights.holder© 2016 Macmillan Publishers Limited, part of Springer Nature. All rights reserved.
dc.identifier.doi10.1038/ng.3699en_US
pubs.author-urlhttp://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000389011100007&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6aen_US
pubs.issue12en_US
pubs.notesNot knownen_US
pubs.publication-statusPublisheden_US
pubs.volume48en_US
qmul.funderCommunication between mother and fetus : Imprinting and endocrine adaptations to pregnancy::Medical Research Councilen_US


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