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dc.contributor.authorVivekananda, Uen_US
dc.contributor.authorNovak, Pen_US
dc.contributor.authorBello, ODen_US
dc.contributor.authorKorchev, YEen_US
dc.contributor.authorKrishnakumar, SSen_US
dc.contributor.authorVolynski, KEen_US
dc.contributor.authorKullmann, DMen_US
dc.date.accessioned2017-03-20T14:12:13Z
dc.date.available2017-01-12en_US
dc.date.issued2017-02-28en_US
dc.date.submitted2017-02-28T18:37:37.396Z
dc.identifier.issn0027-8424en_US
dc.identifier.urihttp://qmro.qmul.ac.uk/xmlui/handle/123456789/21603
dc.format.extent2395 - 2400en_US
dc.relation.ispartofPROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICAen_US
dc.rightsThis is a pre-copyedited, author-produced version of an article accepted for publication in PNAS following peer review. The version of record is available http://www.pnas.org/content/114/9/2395.abstract
dc.subjectchannelopathyen_US
dc.subjectsynaptic transmissionen_US
dc.subjectpotassium channelen_US
dc.titleKv1.1 channelopathy abolishes presynaptic spike width modulation by subthreshold somatic depolarizationen_US
dc.typeArticle
dc.rights.holder© 2017 National Academy of Sciences.
dc.identifier.doi10.1073/pnas.1608763114en_US
pubs.author-urlhttp://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000395101200078&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=612ae0d773dcbdba3046f6df545e9f6aen_US
pubs.issue9en_US
pubs.notesNot knownen_US
pubs.publication-statusPublisheden_US
pubs.volume114en_US


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