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    Author
    Baker, D (107)
    Schmierer, K (59)Giovannoni, G (58)Pryce, G (29)Gnanapavan, S (23)Marta, M (22)Allen-Philbey, K (15)Amor, S (15)Mathews, J (12)Turner, B (11)... View MoreSubjectMultiple sclerosis (18)multiple sclerosis (17)Cladribine (8)Animals (5)autoimmunity (5)Female (5)Multiple Sclerosis (4)CD20 (3)Disease-modifying treatment (3)Encephalomyelitis, Autoimmune, Experimental (3)... View MoreDate Issued2020 - 2022 (31)2011 - 2019 (72)
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    Fingolimod modulates microglial activation to augment markers of remyelination 

    Jackson, SJ; Giovannoni, G; Baker, D (2011-07-05)
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    Brain endothelial miR-146a negatively modulates T-cell adhesion through repressing multiple targets to inhibit NF-κB activation. 

    Wu, D; Cerutti, C; Lopez-Ramirez, MA; Pryce, G; King-Robson, J; Simpson, JE; van der Pol, SM; Hirst, MC; de Vries, HE; Sharrack, B;... (2014-12-17)
    Pro-inflammatory cytokine-induced activation of nuclear factor, NF-κB has an important role in leukocyte adhesion to, and subsequent migration across, brain endothelial cells (BECs), which is crucial for the development ...
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    Brain endothelial miR-146a negatively modulates T-cell adhesion through repressing multiple targets to inhibit NF-kappa B activation 

    Wu, D; Cerutti, C; Lopez-Ramirez, MA; Pryce, G; King-Robson, J; Simpson, JE; van der Pol, SMA; Hirst, MC; de Vries, HE; Sharrack, B;... (2015-03)
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    Setting a research agenda for progressive multiple sclerosis: The International Collaborative on Progressive MS 

    Fox, RJ; Thompson, A; Baker, D; Baneke, P; Brown, D; Browne, P; Chandraratna, D; Ciccarelli, O; Coetzee, T; Comi, G;... (2012-11)
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    Ageing and recurrent episodes of neuroinflammation promote progressive experimental autoimmune encephalomyelitis in Biozzi ABH mice 

    Peferoen, LAN; Breur, M; van de Berg, S; Peferoen-Baert, R; Boddeke, EHWGM; van der Valk, P; Pryce, G; van Noort, JM; Baker, D; Amor, S (2016-10)
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    The problem with repurposing: Is there really an alternative to Big Pharma for developing new drugs for multiple sclerosis? 

    Giovannonin, G; Baker, D; Schmierer, K (2015-01)
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    Genetic Background Can Result in a Marked or Minimal Effect of Gene Knockout (GPR55 and CB2 Receptor) in Experimental Autoimmune Encephalomyelitis Models of Multiple Sclerosis 

    Sisay, S; Pryce, G; Jackson, SJ; Tanner, C; Ross, RA; Michael, GJ; Selwood, DL; Giovannoni, G; Baker, D (2013-10-09)
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    Lesional-targeting of neuroprotection to the inflammatory penumbra in experimental multiple sclerosis 

    Al-Izki, S; Pryce, G; Hankey, DJR; Lidster, K; von Kutzleben, SM; Browne, L; Clutterbuck, L; Posada, C; Chan, AWE; Amor, S;... (2014-01)
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    Neurodegeneration progresses despite complete elimination of clinical relapses in a mouse model of multiple sclerosis. 

    Hampton, DW; Serio, A; Pryce, G; Al-Izki, S; Franklin, RJ; Giovannoni, G; Baker, D; Chandran, S (2013-12-23)
    BACKGROUND: [corrected] Multiple Sclerosis has two clinical phases reflecting distinct but inter-related pathological processes: focal inflammation drives the relapse-remitting stage and neurodegeneration represents the ...
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    Pentraxin-3 is upregulated in the central nervous system during MS and EAE, but does not modulate experimental neurological disease 

    Ummenthum, K; Peferoen, LAN; Finardi, A; Baker, D; Pryce, G; Mantovani, A; Bsibsi, M; Bottazzi, B; Peferoen-Baert, R; van der Valk, P;... (2016-03)
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