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Isolated Addison's disease is unlikely to be caused by mutations in MC2R, MRAP or STAR, three genes responsible for familial glucocorticoid deficiency

dc.contributor.authorDias, RPen_US
dc.contributor.authorChan, LFen_US
dc.contributor.authorMetherell, LAen_US
dc.contributor.authorPearce, SHSen_US
dc.contributor.authorClark, AJLen_US
dc.date.accessioned2016-11-30T13:19:55Z
dc.date.issued2010-02en_US
dc.date.submitted2016-03-01T14:56:17.791Z
dc.identifier.issn0804-4643en_US
dc.identifier.other10.1530/EJE-09-0720
dc.identifier.urihttp://qmro.qmul.ac.uk/xmlui/handle/123456789/18039
dc.description.sponsorshipR P Dias is a Clinical Research Fellow funded by the Medical Research Council.en_US
dc.format.extent357 - 359en_US
dc.language.isoenen_US
dc.relation.ispartofEUR J ENDOCRINOLen_US
dc.rightsCreative Commons Attribution License
dc.subjectACTH RECEPTORen_US
dc.subjectADRENOCORTICOTROPIN RECEPTORen_US
dc.subjectASSOCIATIONen_US
dc.subjectCHILDRENen_US
dc.titleIsolated Addison's disease is unlikely to be caused by mutations in MC2R, MRAP or STAR, three genes responsible for familial glucocorticoid deficiencyen_US
dc.typeArticle
dc.rights.holder2010 European Society of Endocrinology
dc.identifier.doi10.1530/EJE-09-0720en_US
pubs.issue2en_US
pubs.notesNot knownen_US
pubs.volume162en_US


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